How Should I Dose Epi-Androsterone for a First Cycle?

Start with 250–300 mg/day using a cyclodextrin or liposomal oral formulation. Split the dose AM/PM with meals (fat aids absorption). Run for 4 weeks. Most users new to prohormones find this strikes the right balance of strength and hardening without excessive suppression or side effects. | Experience Level | Daily Total | Dosing Schedule | Cycle Length | |-------------------|-------------|-----------------|-------------| | Beginner | 250–300 mg | AM/PM | 4 weeks | | Intermediate | 400–500 mg | AM/PM | 4–6 weeks | | Advanced | 600–750 mg | 2–3× daily | 6–8 weeks | Take each dose with food (preferably containing some fat) to maximize absorption. Avoid raw powder; the poor bioavailability means most will never reach systemic circulation (Leblanc et al., 2003).

What Is the Ideal Timing and Administration Route for Epi-Andro?

Oral is the only practical route — use a branded cyclodextrin or 'Cyclosome' tablet/capsule. BID (morning and evening) dosing is the community standard, since epi-andro's plasma effect lasts roughly 12 hours (half-life: not cleanly characterized; BID is typical in practice). Take each dose with a meal to improve uptake.

Can I Stack Epi-Andro with Other Compounds?

Absolutely — stacking is where epi-andro shines as a lean-hardening base or accent. - With 1-Andro: The community classic for a dry, lean recomp. 1-Andro (1-testosterone precursor) provides mass and aggression; epi-andro sharpens muscle definition and adds strength. - With 4-Andro: Balances a wetter, more estrogenic base with the purely androgenic, dry effect of epi-andro. Common for a "test + DHT" mimic. - With SARM cycles: Epi-andro can be layered in to enhance strength and dryness, but expect HPTA suppression to stack as well. - Contest finishing: Added to injectable cycles for a final hard/dry look without more pinning. No aromatization occurs — stacking with estrogenic compounds is a way to round out the cycle if you find DHT-only runs flatten out your mood, joints, or pumps.

Is There a Loading or Tapering Phase Needed?

No loading or tapering is needed with epi-andro. Start at your target dose day one and hold that for the length of the cycle. For advanced users ramping up to high doses (≥600 mg/day), some will start at 400 mg for the first few days to assess tolerance, but it isn't essential.

What If I Miss a Dose?

Missing a single dose is not catastrophic. Just resume your normal dosing schedule at the next interval — no need to double up. Epi-andro's DHT-conversion window is wide enough that missing an AM or PM dose once in a while will not wreck your progress.

What Post-Cycle Therapy (PCT) Do I Need After Epi-Andro?

A SERM-based PCT is the community gold standard, even at modest doses. Suppression is not as severe as methylated orals, but it is real — LH/FSH and natural T will drop. Standard protocol: - Nolvadex: 20/20/10/10 mg/day each week for 4 weeks or - Clomid: 50/50/25/25 mg/day each week for 4 weeks Also tack on liver, lipid, and mood support as needed (fish oil, citrus bergamot, ashwagandha). Bloodwork on week 2–3 of PCT will show if you've fully recovered or if you need to extend.

What Side Effects Should I Watch Out For, and How Do I Manage Them?

- Androgenic (DHT-driven): Accelerated hair loss, oily skin, increased body/facial hair. If you have MPB genetics, expect hair-shedding unless you aggressively defend with topical RU58841 or pyrilutamide. Oral finasteride/dutasteride will NOT protect you on epi-andro — the DHT is produced downstream (El Kihel, 2012). - Lipid drop: Expect HDL to sink and LDL to rise. Run 500–1000 mg citrus bergamot or consider a statin if you have pre-existing issues. - Suppression: Libido drop and testicular atrophy are both possible by week 3–4 — PCT as above is not optional. - Prostate/urinary: Tadalafil 2.5–5 mg daily covers most issues. Not to be combined with nitrate meds. - Blood pressure: Modest rise is common; monitor weekly, especially if stacking with other orals. - Liver: No significant risk — this is non-methylated (Gupte et al., 2002). - Women: Not recommended (virilization risk is real and not reliably reversible). If any cardiovascular, psychiatric, or urgent endocrine symptoms present, cease immediately and initiate PCT.

Is Epi-Andro Legal, and Will It Show Up on a Drug Test?

Treat epi-andro as a de facto illegal steroid if your job or sport tests for AAS. Epiandrosterone and its metabolites are banned under WADA and show up reliably on urine screens due to their DHT backbone (DEA Final Rule, 2023). While still available OTC in some places, the regulatory risk is rising, and possession/use is at your own discretion. If you are a tested athlete or work in sensitive fields, stay clear.

Epiandrosterone

Epi-Andro · 3β-Androsterone · Isoandrosterone · trans-Androsterone · Stane

Last updated April 18, 2026

Steroids

SteroidProhormone (DHT Precursor)Grey-Marketgrey-market

Best forStrength 5/10

Cycle4–8wk

RiskHigh

43 min read

Half-LifeUnknown

Bioavailability3%

RouteOral

Dose Unitmg

Cycle4–8 weeks

Peak2h

Active Duration12h

MW290.44 g/mol

StorageRoom temp

At a glance

Effectiveness Profile

Strength: 5/10
Muscle Growth: 4/10
Fat Loss: 3/10
Recovery: 3/10
Endurance: 2/10
Cognition: 2/10

Anabolic Rating

Testosterone = 100

Androgenic Rating

Testosterone = 100

Build a stack with Epiandrosterone Predict outcomes ← Compound library

Overview

Epi-andro earned its spot in the grey-market prohormone playbook for one specific reason: it's the cleanest way to run a non-aromatizing DHT precursor without touching a 17α-alkylated oral. Oxidized by 3β-HSD and reduced at the 17-keto, epiandrosterone converts in vivo to dihydrotestosterone — delivering the dry, hard, vascular phenotype that physique-focused users chase on a cut or contest peak, with none of the estrogenic bloat and essentially none of the hepatotoxicity you get from epistane, M1T, or superdrol.

"Epiandrosterone, which is the 5α-reduced DHEA with a 3β-hydroxy-5α-androstan-17-one structure, exhibits androgenic activity after its enzymatic oxidation by 3β-hydroxysteroid dehydrogenase (3β-HSD) and subsequent reduction to DHT." — El Kihel, Steroids (2012)

It's not a mass builder and it shouldn't be treated as one. Scale weight barely moves; the mirror does. The community runs it for a Masteron-lite finish on top of an AAS cycle, as a standalone dry recomp tool for natural users who want something with real teeth, or stacked with 1-Andro or 4-Andro for fully non-aromatizing "wet + dry" runs. Strength goes up, pumps sharpen, and the dry-skin look lands by about week three — provided you're running a properly formulated cyclodextrin or liposomal product, because unformulated raw powder barely clears first-pass metabolism.

The rest of this page covers what you actually need to run it well: dose ladders from first-cycle through advanced, cycle length and stacking (solo, with 1-Andro, with 4-Andro, and as a back-end hardener), PCT protocols, the real side-effect profile (hair, lipids, prostate, BP) and how to mitigate each, plus monitoring bloodwork and the common pitfalls that sink otherwise well-designed cycles.

How Epiandrosterone works

Epi-andro is not itself a meaningful androgen receptor agonist — it's a pro-drug. The whole point of the molecule is that it sits one enzymatic step upstream of 5α-dihydrotestosterone (DHT), and your own liver does the conversion. Everything the compound "does" in the mirror is downstream of that hand-off.

Two-Step Enzymatic Conversion to DHT#

Epiandrosterone is a 5α-reduced, 3β-hydroxy C19 steroid — structurally, DHT with the wrong hydroxyl on the A-ring and the wrong oxidation state at C17. Hepatic 3β-hydroxysteroid dehydrogenase (3β-HSD / 3β-HSOR) oxidizes the 3β-OH to a ketone, producing 5α-androstane-3,17-dione. 17β-HSD then reduces the C17 ketone to a β-hydroxyl, yielding DHT (stanolone).

"Epiandrosterone, which is the 5α-reduced DHEA with a 3β-hydroxy-5α-androstan-17-one structure, exhibits androgenic activity after its enzymatic oxidation by 3β-hydroxysteroid dehydrogenase (3β-HSD) and subsequent reduction to DHT." — El Kihel, L. Steroids, 2012

"It was shown that DHEA and epiandrosterone serve as substrates for human hepatic 3beta-hydroxysteroid oxidoreductase (HSOR), leading to the formation of 5α-androstanedione and ultimately to DHT." — Chalbot S, Morfin R. Drug Metab Dispos, 2005

Practical consequence: because DHT is generated downstream of 5α-reductase, finasteride and dutasteride will not protect your hairline on epi-andro. If you're MPB-prone, topical AR antagonists (RU58841, pyrilutamide) are the only mechanistically sensible scalp defense on cycle.

DHT at the Androgen Receptor — Dry, Hard, No Estrogen#

DHT binds the androgen receptor with roughly three times the affinity of testosterone and dissociates more slowly, producing stronger per-molecule AR signalling in skeletal muscle, CNS, and sebaceous tissue. That drives the usual AR-mediated physique outcomes: increased myofibrillar protein synthesis, amino-acid uptake, satellite cell activation, and strength-per-bodyweight.

The defining feature for physique users is what DHT cannot do: the A-ring is already saturated, so the molecule is not a substrate for aromatase. No estrogen conversion, no water retention, no gyno risk, no E2-driven fat patterning. This is why the subjective feel of an epi-andro run is "harder, drier, tighter" rather than "fuller, heavier, wetter" — you're running a purely androgenic signal with no estrogenic counterweight. It's also why the scale barely moves while the mirror improves: dry lean tissue and reduced subcutaneous water, not glycogen-and-fluid bulk.

Why Oral Bioavailability Is the Whole Game#

Epiandrosterone taken as raw powder is largely wasted. Orally dosed 17-ketosteroids in this family undergo aggressive first-pass sulfation and glucuronidation in the gut wall and liver before they can even reach the 3β-HSD conversion step.

"Oral administration of dehydroepiandrosterone and related androstane derivatives results in extensive first-pass hepatic metabolism with a bioavailability of approximately 3% in primate models." — Leblanc M et al. J Clin Endocrinol Metab, 2003

Practical consequence: the commercial products that work (cyclodextrin-complexed, liposomal, "Cyclosome") aren't marketing gimmicks — the delivery format is doing most of the work. A 500 mg/day dose of unformulated powder delivers a fraction of a 330 mg/day dose of a properly complexed product. Always dose with dietary fat to exploit lymphatic uptake and bypass a portion of first-pass.

Non-Methylated Structure — Minimal Hepatotoxicity#

Unlike true 17α-alkylated orals (superdrol, M1T, epistane, dianabol), epiandrosterone has no C17α methyl group. It doesn't need one — it's not trying to survive first-pass as the active molecule; it's trying to reach the liver and get converted there. That structural choice is the reason AST/ALT typically stay in range on standard doses, and it's the single biggest reason epi-andro gets tolerated for 6–8 week cycles where a real methyl like M1T would be a 2–4 week proposition.

TUDCA at 250–500 mg/day is reasonable precaution, not a mechanistic necessity. The real organ system taking the hit on this compound is your lipid panel, not your hepatocytes.

Off-Target Pharmacology Worth Knowing#

Epiandrosterone has a few non-AR actions that matter at higher doses:

L-type calcium channel blockade in cardiac myocytes — documented in vitro and relevant at supraphysiological exposures.

"Epiandrosterone blocks L-type Ca²⁺ channels in ventricular myocytes at micromolar concentrations, resulting in a negative inotropic effect that could become relevant at supraphysiological exposures." — Gupte SA et al. J Mol Cell Cardiol, 2002

Weak GABA-A negative allosteric modulation — contributes to the mild mood-lift / stimulatory feel some users report, similar to other 3β-hydroxy neurosteroids.
G6PDH / pentose phosphate pathway inhibition — cited historically as the rationale for epiandrosterone's "hypolipidemic" label in early literature, though in practice oral androgen exposure dominates and lipids trend worse on cycle, not better.

Net Practical Picture#

You're taking an oral pro-drug that your liver converts into DHT. You get dry strength, AR-driven hardness, no aromatization, no serious hepatotoxicity, and suppressed HPTA output that needs a proper SERM PCT on the back end. The side-effect profile — HDL drop, accelerated scalp loss in genetically susceptible users, mild BP creep, prostate sensitivity — is the DHT profile, full stop. Manage those on cycle (bergamot/fish oil, topical AR antagonist, daily low-dose tadalafil) and epi-andro is one of the cleaner grey-market tools for a dry recomp or hardening finisher.

Protocol

Level	Dose	Frequency	Notes
Low	250–300 mg	Twice daily	Documented entry-level range
Mid	400–500 mg	Twice daily	Most commonly studied range
High	600–750 mg	Twice daily	Split AM/PM with meals. Advanced users sometimes run 3× daily. Take with dietary fat to improve absorption of the lipophilic steroid.

Cycle length & outcomes

Documented cycle

4–8 weeks

Plateau after

6 wks

Cycle Structure#

Epi-andro is a non-methylated DHT prohormone — no loading phase, no ramp-up, no ester kinetics to manage. It is orally dosed and converted, with the "dry and hard" feel appearing around day 10–14. Because it can't aromatize, there's no wet-to-dry transition mid-cycle and no estrogen rebound at the end. What you do have to plan for is HPTA suppression (mild-to-moderate), lipid drag, and a mandatory SERM PCT.

Run length lives in the 4–8 week window. Under four weeks is barely worth the suppression; past eight, diminishing returns on muscle/strength collide with accelerating HDL drop and hairline anxiety. Six weeks is the sweet spot for most users.

Dose Ladder by Goal#

Goal	Cycle Length	Daily Dose	Split
First cycle / recomp feel-out	4 weeks	250–300 mg	AM + PM with meals
Dry cut / aesthetic hardening	4–6 weeks	400–500 mg	AM + PM with meals
Lean bulk (stacked with 1-andro)	5–6 weeks	300–400 mg	AM + PM with meals
Strength peak / advanced standalone	6–8 weeks	600–750 mg	3× daily with meals
AAS cycle hardening finisher	Final 4 weeks of cycle	500 mg	AM + PM with meals

Dose with dietary fat — epiandrosterone is lipophilic and absorption meaningfully improves alongside a meal containing 15+ g fat. This is the single easiest variable to optimize.

Onset Timing#

Days 1–7: Little to nothing. Some users report a subtle mood/aggression lift by day 4–5 from GABA-A modulation.
Days 7–14: Strength climbs noticeably. Pumps get denser. Skin starts to tighten visually.
Weeks 3–5: Peak effect window — visible hardening, vascularity, a measurable strength bump (community logs run 5–15 lb on main lifts). This is where the mirror changes.
Weeks 6–8: Diminishing returns curve flattens; suppression deepens. Libido and morning wood often dip here.

"Epiandrosterone … exhibits androgenic activity after its enzymatic oxidation by 3β-HSD and subsequent reduction to DHT." — El Kihel, Steroids (2012)

The two-step conversion is why epi-andro is slower-feeling than a true DHT injectable like Masteron — you're waiting on enzymatic throughput, not direct receptor occupancy.

Formulation Matters More Than Dose#

"Oral administration of dehydroepiandrosterone and related androstane derivatives results in extensive first-pass hepatic metabolism with a bioavailability of approximately 3% in primate models." — Leblanc et al., JCEM (2003)

Raw epiandrosterone powder is effectively inert orally. Any real cycle requires a cyclodextrin-complexed, liposomal, or "Cyclosome" formulation — Androvar, Super-DMZ Rx, and similar retail products are built this way for a reason. If you're comparing a 330 mg formulated capsule to 500 mg of bulk powder, the capsule wins easily. This is the single biggest mistake on first cycles.

Bloodwork Cadence#

Timepoint	Panel
Pre-cycle baseline	Total/free T, LH, FSH, E2, full lipids, AST/ALT, CBC, BP
Week 3–4 (mid-cycle)	Lipids, AST/ALT, BP (home cuff)
End of cycle → PCT start	Total T, LH, FSH
Week 10–12 (post-PCT)	Total T, LH, FSH, lipids — confirm recovery

Non-methylated status keeps liver values boringly in-range for most users, but lipids will move — expect HDL to drop 10–25% and LDL to nudge up. Citrus bergamot 500–1000 mg/day and 2–4 g fish oil daily take the edge off; back-to-back cycles without a lipid recovery window are the fastest way to earn a cardiology referral in your 40s.

PCT Timing#

Start SERM PCT the day after your last dose — no clearance gap needed given epi-andro's short functional duration.

Nolvadex: 20/20/10/10 mg over 4 weeks (preferred for most users — kinder on lipids than clomid)
Clomid: 50/50/25/25 mg over 4 weeks (alternative)

Natty support (tongkat, ashwagandha, DAA) can ride alongside but is not a replacement. Running a 500 mg × 6 week cycle without a SERM and hoping you "bounce back" is how people end up on TRT at 32.

Time Off#

Minimum time-off = time-on + PCT length. A 6-week cycle + 4-week PCT means at least 10 weeks clean before the next run. Lipids and HPTA both want that window. Two to three cycles per year is the sustainable ceiling — more than that and you're quietly turning a prohormone habit into a low-dose oral AAS habit with worse pharmacokinetics.

Risks & mistakes

Common (most users)#

Oily skin and acne — DHT is doing what DHT does. Daily salicylic acid cleanser, niacinamide serum, and keeping dietary dairy/sugar in check handle most of it. If it's escalating to cystic, the dose is too high or the individual's genetics may not tolerate DHT loads.
Accelerated scalp hair shedding (if MPB-prone) — this is the single most common reason users quit epi-andro. Oral 5AR inhibitors will not save you here — the DHT is delivered downstream of 5α-reductase. Topical RU58841 or pyrilutamide (AR antagonists at the follicle) are the only mechanistically valid scalp defenses during a cycle. If a receding hairline is already present, choose an alternative compound.
Libido swings — strong libido and morning wood for the first 2–3 weeks, then a dropoff as HPTA suppression kicks in. Daily tadalafil 2.5–5 mg smooths this out and pulls double duty for BP and prostate.
Aggression / assertiveness uptick — typical DHT tone. Usually wanted. Keep it out of the cage in week 3–4 when sleep may also be lighter.
Mild BP rise — cuff at home 2–3×/week. Citrus bergamot, cardio 3×/week, and sodium discipline keep it in check. If resting is pushing past 135/85, back down.
"Flat" look for the first 7–10 days — epi-andro doesn't aromatize, so you lose the estrogenic water softness early in the cycle before the DHT hardening kicks in. Don't panic-dose higher.

Uncommon (dose-dependent or individual)#

Lipid drag — HDL down, LDL up. Predictable for any oral non-aromatizing androgen, even without 17α-alkylation. Get a baseline panel pre-cycle and recheck at week 3–4. If HDL drops below 35 mg/dL or LDL goes past 160, cut the dose or end the cycle early. Citrus bergamot 500–1000 mg/day and fish oil 2–4 g/day are the standard blunting stack.
Prostate fullness / frequent urination — DHT-mediated prostatic growth. Daily tadalafil 2.5–5 mg resolves this for most users. If it persists or there's hesitancy/incomplete voiding, pull the cycle.
Mood / sleep disruption at higher doses (600–750 mg/day) — light sleep, waking hot, increased irritability. Drop to 400–500 mg/day.
Suppressed morning wood by week 4–5 — expected signal that LH/FSH are down. Not a problem during the cycle; it's the reason PCT is non-negotiable.
Gut / GI upset — usually the formulation, not the molecule. Take with meals containing fat.

Rare but serious#

Hypertensive crisis — anyone running epi-andro on top of untreated hypertension is courting this. Warning signs: headache that won't quit, visual disturbance, resting BP >160/100. Stop immediately.
Cardiac symptoms at supraphysiological exposures — high-dose in vitro work shows L-type Ca²⁺ channel blockade and negative inotropy in ventricular myocytes.

"Epiandrosterone blocks L-type Ca2+ channels in ventricular myocytes at micromolar concentrations, resulting in a negative inotropic effect that could become relevant at supraphysiological exposures." — Gupte et al., J Mol Cell Cardiol (2002)

Clinically relevant only at abusive doses, but it's the mechanistic reason mega-dosing this compound is a bad idea.

Persistent HPTA suppression past a standard 4-week PCT — uncommon at 4–6 week cycle lengths, more likely with 8-week runs or back-to-back cycles without full recovery. Bloodwork (LH, FSH, total T) 6–8 weeks post-PCT is how you catch it. If T is still suppressed, you need an endocrinology workup, not another PCT round.
Severe acne / scarring breakouts — in genetically susceptible users, epi-andro can trigger the kind of cystic acne that needs isotretinoin to resolve. If the first 2 weeks show aggressive cystic activity, end the cycle.

Hard contraindications#

Active or prior prostate cancer or hormone-sensitive cancer — DHT drives growth. Non-negotiable.
Untreated hypertension or dyslipidemia — control these before you start an oral androgen, not while you're running one.
Planned conception within the next 6 months — HPTA suppression hits LH/FSH and semen parameters. Finish a cycle, run full PCT, recheck bloodwork, then try.
Pregnancy or possibility of pregnancy — androgens are teratogenic to a female fetus. Do not handle or dose.
Female use — epi-andro converts to DHT in vivo. Virilization (voice deepening, clitoral enlargement, androgenic hair pattern changes) is real and past a threshold is not reversible. Women looking for recomp have better tools.
Tested athletes — WADA-banned and detectable on standard urinary androgen-metabolite panels via epiandrosterone-glucuronide. There is no "clean" window.
Advanced male pattern baldness where hair retention matters to you — this is a DHT delivery vehicle. That's the entire mechanism. Pick a different compound.

Gender & PCT considerations#

Women: not recommended at any dose. The DHT conversion pathway is the whole point of the compound, and the virilization risk it carries (voice, clitoral tissue, androgenic hair pattern) has a partial-irreversibility threshold that sneaks up on users. Anavar or low-dose primobolan are the conventional female-tolerable picks — epi-andro isn't.

Men — PCT: run it, even though the marketing calls this a "mild" prohormone. Bloodwork from 6-week users consistently shows meaningful LH/FSH drops.

Cycle length	PCT protocol
4 weeks, ≤400 mg/day	Nolvadex 20/20/10/10 (4 weeks)
6 weeks, 400–500 mg/day	Nolvadex 20/20/20/10 or Clomid 50/50/25/25 (4 weeks)
8 weeks or 600+ mg/day	Clomid 50/50/25/25 + Nolvadex 20/20/20/20 (4 weeks)

Support: natural test-support stack (DAA, tongkat, ashwagandha) layered on top is fine but not a substitute for a SERM. Recheck total T, LH, and FSH 4 weeks after PCT ends to confirm recovery before planning the next cycle. Give lipids 6–8 weeks clean between runs — don't blast-and-cruise a prohormone.

Stack & combine

Pairwise synergies

Multipliers applied when these compounds run together. Values > 1 indicate a bonus on that axis. Tap a partner to expand the mechanism.

Partner	Type	Lean	Fat loss	Recovery
TUDCA	synergistic	×1.03	×1.00	×1.15

FAQ — Epiandrosterone

PCT & ancillaries

These are separate products from our affiliate partners, not Epiandrosterone offers. Their inclusion does not mean they are required or recommended for use.

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Research & citations

5 studies cited on this page.

Conclusion

Epi-andro is the no-nonsense choice for dry strength, hardening, and vascularity when you want a DHT-driven look without managing estrogen.

Key takeaways:

Standard cycle: 400–500 mg/day oral (cyclodextrin/liposomal) split AM/PM, 4–6 weeks
Bioavailability is poor unless you use a formulated (not raw powder) product
Stacks well with 1-Andro or 4-Andro for lean or balanced cycles
Does not aromatize — expect dry muscle, strength, and visible muscle separation, not big scale weight jumps
Full SERM-based PCT is required — think nolvadex 20/20/10/10 or clomid 50/50/25/25
Real DHT-driven side effects: MPB acceleration and mild HDL suppression are the big flags; topical AR blockers help hair, citrus bergamot for lipids

If you want a reliable, low-bloat hardening prohormone with clear lines of strength and muscle detail, epi-andro is one of the most user-proven options in the category. Use it intelligently and you get a dry, dense look without the legal heat or liver hit of harsher orals.

Similar compounds

Testosterone

Testosterone is the compound every other anabolic is measured against — literally. Its anabolic:androgenic ratio of 100:100 is the reference point the entire AA

Muscle 9/10A:100 · An:100SubQ12–16wkPCT req.

Sustanon 250

Why Sustanon 250 Earned Its Place Sustanon 250 is the four-ester testosterone blend that built half the physiques in European gyms. One ampoule delivers 30mg

Muscle 9/10A:100 · An:100IM12–16wkPCT req.

Nandrolone

Why Nandrolone Still Anchors Serious Mass Cycles Nandrolone is one of the oldest compounds in the bodybuilding playbook for a reason: it builds thick, full mu

Recovery 9/10A:125 · An:37IM12–16wkPCT req.

Trenbolone

Why Trenbolone Has the Reputation It Does Trenbolone is the most potent anabolic androgen in practical use — a 19-nor compound with roughly 3–5× the androgen

Muscle 9/10A:500 · An:500IM6–12wkPCT req.

Trestolone

Trestolone — MENT, the 7α-methyl-19-nortestosterone molecule the Population Council originally designed as a male contraceptive — has quietly become one of the

Muscle 9/10A:600 · An:200SubQ6–10wkPCT req.

Dihydroboldenone

Why DHB Has a Cult Following Dihydroboldenone — sold as 1-Test Cypionate or 1-Test Acetate — is the 5α-reduced analog of boldenone, structurally a DHT molecul

Muscle 7/10A:200 · An:100IM10–16wkPCT req.

Comparisons

Epiandrosterone

1-Andro

Disclosures

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This page is for informational and educational purposes only. Epiandrosterone is a research compound intended for laboratory research use only. Consult a qualified medical professional before relying on any compound described here. This is not medical advice.