Comparison

Trevogrumab vs Follistatin-344

Systemic muscle-sparing vs. site-specific myostatin inhibition — actual trial data versus experimental niche.

Effectiveness Profile

Trevogrumab
Follistatin-344

At a Glance

 TrevogrumabFollistatin-344
TypeOtherOther
Legal statusResearchResearch
Half-life~2–4 weeks (IgG4 class estimate)~1 hour (circulating); longer tissue residence at depot site
Preferred routeIVSubQ (site-specific over target muscle)
Dose frequencycustomonce-daily
Beginner dose200–200 mg50–100 mcg
Intermediate dose400–400 mg100–200 mcg
Advanced dose400–400 mg300–500 mcg
Cycle length26–52 wks2–4 wks
Bioavailability100%
Time to peak2h0.25h
Active duration672h24h
Storage2–8°C refrigerated; do not freeze; protect from lightLyophilized: -20°C long-term, 2–8°C short-term. Reconstituted: 2–8°C refrigerated, use within 2–3 weeks.
PCT requiredNoNo
Ancillaries requiredNoNo
Safe for womenYesYes

Verdict

Trevogrumab wins for legitimate systemic muscle-sparing with modern weight-loss agents (especially GLP-1s), documented efficacy in human-grade trials, a clean selectivity profile with minimal off-target pathway impact, and no direct risk of local tissue or vision complications.

Follistatin-344 wins for site-targeted hypertrophy on lagging muscles, direct tissue-level application, broader inhibition (myostatin, activin, TGF-β ligands), off-label blendability in research stacks, and practical accessibility from peptide sources, despite a lack of human data for physique outcomes.

Pick A or B?

Pick Trevogrumab if:

  • The protocol is GLP-1–based (semaglutide/tirzepatide) and requires preservation of lean mass during aggressive body recomposition.
  • Documented, peer-reviewed efficacy data is a primary driver in compound selection.
  • Avoidance of off-target pathway inhibition is critical (clean avoidance of activin A/GDF11 actions).
  • Sourcing from legitimate clinical trials or collaborations is possible.
  • Seeking a system-wide muscle-sparing adjunct, not localized muscle growth.

Pick Follistatin-344 if:

  • The primary goal is hypertrophy focused on a stubborn or lagging muscle group (site-specific application).
  • Research requires a broad-spectrum ligand trap (myostatin, activin, TGF-β trapping) not achievable with antibody-only approaches.
  • Rapid access to experimental peptides is needed (peptide-market sourcing, not limited by clinical pipeline).
  • Willingness to operate in an under-documented, "grey area" research domain.
  • Stacking with IGF-1 LR3, MGF, or GH/GHRPs for synergistic hypertrophy at the tissue level is planned.

Where to Buy

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